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Health & Fitness

Redox Signaling Found Critical to Curbing Cell Suicide!

Researchers at University of Vermont College of Medicine have found that Redox Signaling molecules are crucial in the regulation of apoptosis.

The study findings, published in volume 16 issue 6 of the Journal of Antioxidants and Redox Signaling state, “Redox-based signaling governs a number of important pathways in tissue homeostasis. Among the biological processes regulated by redox signaling, apoptosis or programmed cell death is a highly conserved process important for tissue homeostasis.”

The study further stated, “Apoptosis can be triggered by a wide variety of stimuli, including death receptor ligands, environmental agents, and cytotoxic drugs. Recent discoveries demonstrate that redox-based changes are required for efficient activation of apoptosis. Among these redox changes, alterations in the abundant thiol, glutathione (GSH), and the oxidative post-translational modification, protein S-glutathionylation (PSSG) have come to the forefront as critical regulators of apoptosis.”

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“This is an important finding to support the importance of Redox Signaling Molecules. Apoptosis, a foundational cellular mechanism, is preserved at all cost,” said Robertson Ward, M.D., FAAFP, member of the ASEA Science Advisory Council.

ASEA, LLC is a Salt Lake City-based company whose flagship product, also called ASEA, is the world’s first and only source of stable, balanced, native-to-the-body Redox Signaling molecules.

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Redox Signaling Molecules are produced naturally by the mitochondria in every cell of our body. But as people age they produce less and less of these molecules, which are vitally important to activating antioxidants, promoting cellular health, and enhancing cellular communication.

ASEA is the only method to re-introduce Redox Signaling molecules into the human body. It is sold in a 32-ounce bottle and an eight-ounce flexible pouch made from environmentally friendly materials. The product can be purchased through ASEA’s wide network of independent associates.

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